Publications

Vahlas Z et al. Life Science Alliance (2026), doi: https://doi.org/10.26508/lsa.202503333 — Glycolysis inhibition in TB-driven metabolic rewiring reduces HIV-1 spread in macrophages.

Deyts C et al. EBioMedicine (2026), doi: https://doi.org/10.1016/j.ebiom.2025.106077 — Alveolar macrophage dysfunction as an early determinant of tuberculosis susceptibility in type 2 diabetes. (Commentary)

Bomfim CC et al. Microbiology Spectrum (2026, in press) — Dual human lung models reveal compartment-specific activity of anti-tuberculosis drugs and host-directed therapies.

Rivault A et al. PLoS Pathogens (2025), doi: https://doi.org/10.1371/journal.ppat.1013183 — HIV-1 Tat promotes pathogen replication by inhibiting autophagy and endocytosis.

Dupuy P et al. Cold Spring Harbor Perspectives in Medicine (2025), doi: https://doi.org/10.1101/cshperspect.a041819 — From phagosomes to niches: macrophage biology in tuberculosis revisited. (Book chapter)

Monard SC et al. bioRxiv (2025), doi: https://doi.org/10.1101/2025.03.04.641465 — Identification of a unique TUBB3+ cell population in the tuberculosis granuloma.

Barros J et al. bioRxiv (2025), doi: https://doi.org/10.1101/2025.07.07.663524 — Glycolysis inhibition reduces HIV-1 spread in TB-conditioned macrophages.

Blot C et al. Cell Reports (2024), doi: https://doi.org/10.1016/j.celrep.2024.114720 — Leishmania exploits Nrf2-dependent anti-ferroptosis pathways to escape macrophage death.

Maio M et al. eLife (2024), doi: https://doi.org/10.7554/eLife.89319 — Elevated glycolysis limits HIF1A-driven dendritic cell migration in tuberculosis.

Leon-Icaza SA et al. PLoS Pathogens (2023), doi: https://doi.org/10.1371/journal.ppat.1011559 — Druggable redox pathways in Mycobacterium abscessus using patient-derived airway organoids.

Dupont M et al. Journal of Leukocyte Biology (2022), doi: https://doi.org/10.1002/JLB.4MA0422-730R — IFN-I dysregulation by M. tuberculosis exacerbates HIV-1 infection in macrophages.

Valtierra-Alvarado MA et al. Human Immunology (2022), doi: https://doi.org/10.1016/j.humimm.2022.08.011 — Increased CD14+HLA-DRlow cells in poorly controlled type 2 diabetes.

Benet S et al. Journal of Extracellular Vesicles (2021), doi: https://doi.org/10.1002/jev2.12046 — SIGLEC-1 variant limits antigen exchange and dissemination of Mycobacterium tuberculosis.

Valtierra-Alvarado MA et al. Immunology & Cell Biology (2021), doi: https://doi.org/10.1111/imcb.12497 — Type 2 diabetes alters macrophage-mediated regulation of monocyte differentiation.

Bernard-Raichon L et al. Journal of Immunology (2021), doi: https://doi.org/10.4049/jimmunol.2001044 — Commensal bacteria induce regulatory T cells and reduce TB-associated lung inflammation.

Bagayoko S et al. PLoS Pathogens (2021), doi: https://doi.org/10.1371/journal.ppat.1009927 — Lipid peroxidation drives ExoU-mediated necrosis in Pseudomonas aeruginosa.

Iakobachvili N et al. Molecular Microbiology (2021), doi: https://doi.org/10.1111/mmi.14824 — Mycobacteria interactions in human airway organoids.

Pires D et al. Frontiers in Immunology (2021), doi: https://doi.org/10.3389/fimmu.2021.742822 — Cystatin C modulation enhances anti-mycobacterial responses.

Dupont Mψ et al. eLife (2020), doi: https://doi.org/10.7554/eLife.52535 — TB-associated IFN-I induces Siglec-1 and promotes HIV-1 spread via nanotubes.

Valtierra-Alvarado MA et al. Journal of Diabetes Complications (2020), doi: https://doi.org/10.1016/j.jdiacomp.2020.107708 — Metabolic control in diabetes shapes macrophage phenotype.

Genoula Mψ et al. PLoS Pathogens (2020), doi: https://doi.org/10.1371/journal.ppat.1008929 — Mtb rewires macrophage metabolism to promote foam cell formation.

Marín Franco JLψ et al. Cell Reports (2020), doi: https://doi.org/10.1016/j.celrep.2020.108547 — Host-derived lipids impair macrophage function via HIF-1α.

Dupont M et al. Médecine/Sciences (2020), doi: https://doi.org/10.1051/medsci/2020155 — Siglec-1/CD169 as a catalyst of TB–HIV synergy. (Commentary)